Koch / Chrousos Endocrine Hypertension

Underlying Mechanisms and Therapy
1. Auflage 2012
ISBN: 978-1-60761-548-4
Verlag: Humana Press
Format: PDF
Kopierschutz: 1 - PDF Watermark

E-Book, Englisch, 318 Seiten, eBook

Reihe: Contemporary Endocrinology

The prevalence of hypertension is almost three times as high as that of diabetes mellitus type 2, with both conditions being major risk factors for stroke, ischemic heart disease, cardiac arrhythmias, and heart failure.  The exact prevalence of hypertension related to hormonal derangements (endocrine hypertension) is not known but estimated to affect less than 15% of hypertensive patients.  Recent scientific discoveries have increased the understanding of the pathophysiologic mechanisms of hypertension.  In Endocrine Hypertension, a renowned panel of experts provides a comprehensive, state-of-the-art overview of this disorder, discussing when to assign an endocrine cause in one of  many conditions that may present with hypertension.   The first part of Endocrine Hypertension is dedicated to adrenal causes.  The second part of the volume concerns potential nonadrenal causes of hypertension, such as growth hormone excess or deficiency, primary hyperparathyroidism, vitamin D deficiency, testosterone deficiency, insulin resistance, obesity-associated hypertension, and the role of central mineralocorticoid receptors and cardiovascular disease. An important contribution to the literature, Endocrine Hypertension is an indispensable reference not only for endocrinologists, diabetologists, and adrenal investigators, but also for translational scientists and clinicians from cardiology, internal medicine, pediatrics, family medicine, geriatrics, urology, and reproductive medicine / gynecology.

Zielgruppe

Professional/practitioner

Autoren/Hrsg.

Koch, Christian A

Chrousos, George P

Inhaltsverzeichnis

Primary Aldosteronism: Progress in Diagnosis, Therapy, and Genetics.-Syndromes of Mineralocorticoid Excess.- Hypertension in Patients with Cushing’s Syndrome.- Primary Generalized Familial and Sporadic Glucocorticoid Resistance (Chrousos syndrome) and Hypersensitivity.- Congenital Adrenal Hyperplasia.- Adrenal Incidentalomas and Adrenal Hypertension.- Pheochromocytoma: Unmasking the Chameleon.- Hypertension in Growth hormone excess and deficiency.- Primary Hyperparathyroidism and Hypertension.- Hypertension, Vitamin D deficiency, and Calcium Metabolism.- Testosterone Deficiency or Male Hypogonadism.- Insulin Resistance and Hypertension.- Obesity-associated Hypertension.-Central mineralocorticoid receptors and cardiovascular disease.     Primary Aldosteronism: Progress in Diagnosis, Therapy, and Genetics.-Syndromes of Mineralocorticoid Excess.- Hypertension in Patients with Cushing’s Syndrome.- Primary Generalized Familial and Sporadic Glucocorticoid Resistance (Chrousos syndrome) and Hypersensitivity.- Congenital Adrenal Hyperplasia.- Adrenal Incidentalomas and Adrenal Hypertension.- Pheochromocytoma: Unmasking the Chameleon.- Hypertension in Growth hormone excess and deficiency.- Primary Hyperparathyroidism and Hypertension.- Hypertension, Vitamin D deficiency, and Calcium Metabolism.- Testosterone Deficiency or Male Hypogonadism.- Insulin Resistance and Hypertension.- Obesity-associated Hypertension.-Central mineralocorticoid receptors and cardiovascular disease.     Primary Aldosteronism: Progress in Diagnosis, Therapy, and Genetics.-Syndromes of Mineralocorticoid Excess.- Hypertension in Patients with Cushing’s Syndrome.- Primary GeneralizedFamilial and Sporadic Glucocorticoid Resistance (Chrousos syndrome) and Hypersensitivity.- Congenital Adrenal Hyperplasia.- Adrenal Incidentalomas and Adrenal Hypertension.- Pheochromocytoma: Unmasking the Chameleon.- Hypertension in Growth hormone excess and deficiency.- Primary Hyperparathyroidism and Hypertension.- Hypertension, Vitamin D deficiency, and Calcium Metabolism.- Testosterone Deficiency or Male Hypogonadism.- Insulin Resistance and Hypertension.- Obesity-associated Hypertension.-Central mineralocorticoid receptors and cardiovascular disease.